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  • Varicella Zoster Virus Infection: Clinical Features . . .
    Zoster sine herpete (pain without rash) is caused by reactivation of VZV , a concept first supported by the description of dermatomal distribution radicular pain in areas distinct from pain with rash in zoster patients Currently, most clinicians regard zoster sine herpete exclusively as the rare occurrence of chronic radicular pain without
  • VZV: pathogenesis and the disease consequences of primary . . .
    Severe abdominal or back pain is a clinical sign of serious primary VZV infection in high-risk patients but the etiology of the symptoms is unknown; it is possible that it is related to early infection of sensory ganglia by hematogenous spread of the virus
  • Shingles (herpes zoster) - World Health Organization (WHO)
    Shingles, also known as herpes zoster, is a viral infection that causes a painful rash It is caused by the varicella-zoster virus (VZV), the same virus that causes chickenpox After a person recovers from chickenpox, the virus remains inactive in the body
  • Herpes zoster infection - The BMJ
    Herpes zoster is caused by reactivation of a primary infection with varicella zoster virus 1 After a primary infection, the virus lies dormant in dorsal root or cranial nerve ganglia Reactivation causes the typical dermatomal pain and vesicular rash (fig 1)
  • VZV: pathogenesis and the disease consequences of primary . . .
    The tropism of VZV for skin is the most obvious clinical manifestation of VZV infection, producing the vesicular cutaneous lesions that are associated with varicella and zoster The site of initial VZV infection in naïve hosts is thought to be mucosal epithelial cells of the upper respiratory tract
  • Varicella Zoster Viral Disease - Mayo Clinic Proceedings
    Varicella zoster viras (VZV) is the etiologic agent of 2 common diseases: varicella (chickenpox) and herpes zoster (HZ) 1 In 1875, varicella was found to be an infectious disease by inoculation of volunteers with vesicle fluid from a person with varicella 2 A study in 1892 demonstrated that children exposed to people with HZ could develop
  • The Biology of Varicella-Zoster Virus Replication in the Skin
    When reactivated, VZV travels within the axon in anterograde manner to reach the innervated dermatome where it causes herpes zoster (HZ) or shingles, characterised by a localised painful vesicular rash [4, 5]





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