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  • APOE Models - The Jackson Laboratory
    A 30kB deletion including mouse Apoe to Apoc2 is replaced by the syntenic 47kB region from human chromosome 19 including human APOE (isoform E4) to APOC APOE Models offered by the Model-AD Consortium
  • The Apoe− − mouse model: a suitable model to study cardiovascular and . . .
    This review aims to highlight the usefulness of Apoe − − mouse model in the context of inhalation toxicology studies addressing questions related to the role of cigarette smoke (CS), cessation, and candidate modified risk tobacco products (cMRTPs) in the development of atherosclerosis and pulmonary diseases (Fig 1)
  • Astrocytic APOE4 removal confers cerebrovascular protection despite . . .
    This current study identified that in a mouse model of mixed CAA plaque pathology, removal of a major source of APOE produced by astrocytes increased CAA but provided protection by reducing neuritic dystrophy and ameliorating CAA-induced vascular damage
  • Animal models of cerebral amyloid angiopathy - PubMed
    In the past two decades, a variety of transgenic mouse models expressing the human Aβ precursor protein (APP) has been developed Many of these mouse models develop CAA in addition to senile plaques, whereas some of these models were generated specifically to study CAA
  • B6J-Apoe KO Mice | Metabolic Disease Models | Research Models . . . - Cyagen
    The B6J-Apoe KO mouse is a model of ApoE deficiency It was generated by gene editing technology to knock out the Apoe gene in mice ApoE protein synthesis is blocked in these mice, leading to elevated cholesterol levels and spontaneous atherosclerosis
  • Independent APOE4 knock-in mouse models display reduced brain APOE . . .
    We compared phenotypes associated with APOE4 in two targeted replacement APOE mouse model, APOE -TR and JAX-APOE mice APOE4 genotype results in reduced APOE protein levels, altered inflammatory markers at baseline, and simplified dendritic spine density compared to APOE3 across model
  • APOE from patient-derived astrocytic extracellular vesicles . . . - AAAS
    APOE attenuated microglial reactivity, neuroinflammation, and brain lesion in a mouse model of NMOSD, and genetic knockdown of LRP1 could block these effects ADEVs from patients had a slightly stronger inhibitory effect on brain lesions than ADEVs from HCs 7 days after NMOSD induction
  • The Apoe- - mouse model: a suitable model to study cardiovascular and . . .
    Atherosclerosis-prone apolipoprotein E-deficient (Apoe− −) mice display poor lipoprotein clearance with subsequent accumulation of cholesterol ester-enriched particles in the blood, which promote the development of atherosclerotic plaques Therefore, the Apoe− − mouse model is well established for the study of human atherosclerosis The
  • The Apoe(- -) mouse model: a suitable model to study . . . - PubMed
    Atherosclerosis-prone apolipoprotein E-deficient (Apoe(- -)) mice display poor lipoprotein clearance with subsequent accumulation of cholesterol ester-enriched particles in the blood, which promote the development of atherosclerotic plaques





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